Nothing Special   »   [go: up one dir, main page]

Christaki et al., 2014 - Google Patents

The complex pathogenesis of bacteremia: from antimicrobial clearance mechanisms to the genetic background of the host

Christaki et al., 2014

View PDF @Full View
Document ID
3860423350073963383
Author
Christaki E
Giamarellos-Bourboulis E
Publication year
Publication venue
Virulence

External Links

Snippet

Bacteremia develops when bacteria manage to escape the host immune mechanisms or when the otherwise well-orchestrated immune response fails to control bacterial spread due to inherent or acquired immune defects that are associated with susceptibility to infection …
Continue reading at www.tandfonline.com (PDF) (other versions)

Similar Documents

Publication Publication Date Title
Christaki et al. The complex pathogenesis of bacteremia: from antimicrobial clearance mechanisms to the genetic background of the host
Tuffs et al. Manipulation of innate and adaptive immunity by staphylococcal superantigens
Teng et al. Neutrophils and immunity: from bactericidal action to being conquered
Lei-Leston et al. Epithelial cell inflammasomes in intestinal immunity and inflammation
Becker et al. Pattern recognition pathways leading to a Th2 cytokine bias in allergic bronchopulmonary aspergillosis patients
Peloquin et al. Mechanisms of pediatric inflammatory bowel disease
Bachert et al. Pathophysiology of chronic rhinosinusitis, pharmaceutical therapy options
Lappegård et al. Human genetic deficiencies reveal the roles of complement in the inflammatory network: lessons from nature
Cho et al. Calprotectin increases the activity of the SaeRS two component system and murine mortality during Staphylococcus aureus infections
Hebecker et al. Pathogenicity mechanisms and host response during oral Candida albicans infections
Wang et al. Dectin-3 deficiency promotes colitis development due to impaired antifungal innate immune responses in the gut
Hasegawa et al. Nucleotide-binding oligomerization domain 1 mediates recognition of Clostridium difficile and induces neutrophil recruitment and protection against the pathogen
Xu et al. Streptococcal co‐infection augments C andida pathogenicity by amplifying the mucosal inflammatory response
Nguyen et al. Autophagy and Crohn's disease
Bestebroer et al. Functional basis for complement evasion by staphylococcal superantigen‐like 7
Greenlee-Wacker et al. Phagocytosis of Staphylococcus aureus by human neutrophils prevents macrophage efferocytosis and induces programmed necrosis
Yuan et al. Clinical relevance of TLR2, TLR4, CD14 and FcγRIIA gene polymorphisms in Streptococcus pneumoniae infection
Rabes et al. The C-type lectin receptor Mincle binds to Streptococcus pneumoniae but plays a limited role in the anti-pneumococcal innate immune response
Herzog et al. High nuclease activity of long persisting Staphylococcus aureus isolates within the airways of cystic fibrosis patients protects against NET-mediated killing
Zheng et al. The role of pattern recognition receptors in the innate recognition of Candida albicans
Keita et al. Barrier dysfunction and bacterial uptake in the follicle‐associated epithelium of ileal Crohn's disease
Rabes et al. Inflammasomes in pneumococcal infection: innate immune sensing and bacterial evasion strategies
Shaghayegh et al. Chronic rhinosinusitis, S. aureus biofilm and secreted products, inflammatory responses, and disease severity
Levy et al. Inflammasomes and the microbiota—partners in the preservation of mucosal homeostasis
Lorenz et al. Full functional activity of SSL7 requires binding of both complement C5 and IgA