Nothing Special   »   [go: up one dir, main page]

Blufstein et al., 2018 - Google Patents

Human periodontal ligament cells exhibit no endotoxin tolerance upon stimulation with Porphyromonas gingivalis lipopolysaccharide

Blufstein et al., 2018

View PDF
Document ID
7789125263615623568
Author
Blufstein A
Behm C
Nguyen P
Rausch‐Fan X
Andrukhov O
Publication year
Publication venue
Journal of Periodontal Research

External Links

Snippet

Background/Objectives Endotoxin tolerance is characterized by a state of hyporesponsiveness after confrontation with endotoxins such as lipopolysaccharides (LPS) at low concentrations. The aim of this study was to investigate, whether pretreatment with …
Continue reading at onlinelibrary.wiley.com (PDF) (other versions)

Classifications

    • G—PHYSICS
    • G01—MEASURING; TESTING
    • G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
    • G01N33/00—Investigating or analysing materials by specific methods not covered by the preceding groups
    • G01N33/48—Investigating or analysing materials by specific methods not covered by the preceding groups biological material, e.g. blood, urine; Haemocytometers
    • G01N33/50—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing
    • G01N33/68—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving proteins, peptides or amino acids
    • G01N33/6893—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving proteins, peptides or amino acids related to diseases not provided for elsewhere
    • G—PHYSICS
    • G01—MEASURING; TESTING
    • G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
    • G01N33/00—Investigating or analysing materials by specific methods not covered by the preceding groups
    • G01N33/48—Investigating or analysing materials by specific methods not covered by the preceding groups biological material, e.g. blood, urine; Haemocytometers
    • G01N33/50—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing
    • G01N33/53—Immunoassay; Biospecific binding assay
    • G01N33/569—Immunoassay; Biospecific binding assay for micro-organisms, e.g. protozoa, bacteria, viruses
    • G—PHYSICS
    • G01—MEASURING; TESTING
    • G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
    • G01N33/00—Investigating or analysing materials by specific methods not covered by the preceding groups
    • G01N33/48—Investigating or analysing materials by specific methods not covered by the preceding groups biological material, e.g. blood, urine; Haemocytometers
    • G01N33/50—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing
    • G01N33/5005—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving human or animal cells
    • G01N33/5008—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving human or animal cells for testing or evaluating the effect of chemical or biological compounds, e.g. drugs, cosmetics
    • G—PHYSICS
    • G01—MEASURING; TESTING
    • G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
    • G01N2333/00—Assays involving biological materials from specific organisms or of a specific nature
    • G01N2333/435—Assays involving biological materials from specific organisms or of a specific nature from animals; from humans
    • G01N2333/46—Assays involving biological materials from specific organisms or of a specific nature from animals; from humans from vertebrates
    • G01N2333/47—Assays involving proteins of known structure or function as defined in the subgroups
    • G01N2333/4701—G01N2333/4701
    • G—PHYSICS
    • G01—MEASURING; TESTING
    • G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
    • G01N2800/00—Detection or diagnosis of diseases
    • G01N2800/28—Neurological disorders

Similar Documents

Publication Publication Date Title
Blufstein et al. Human periodontal ligament cells exhibit no endotoxin tolerance upon stimulation with Porphyromonas gingivalis lipopolysaccharide
Wara‐aswapati et al. RANKL upregulation associated with periodontitis and Porphyromonas gingivalis
Tofiño-Vian et al. Extracellular vesicles from adipose‐derived mesenchymal stem cells downregulate senescence features in osteoarthritic osteoblasts
Zhao et al. Wnt5a promotes inflammatory responses via nuclear factor κB (NF-κB) and mitogen-activated protein kinase (MAPK) pathways in human dental pulp cells
Farez et al. Toll-like receptor 2 and poly (ADP-ribose) polymerase 1 promote central nervous system neuroinflammation in progressive EAE
Epron et al. Monocytes and T cells cooperate to favor normal and follicular lymphoma B-cell growth: role of IL-15 and CD40L signaling
Fukada et al. Factors involved in the T helper type 1 and type 2 cell commitment and osteoclast regulation in inflammatory apical diseases
Andrukhov et al. Soluble CD14 enhances the response of periodontal ligament stem cells to P. gingivalis lipopolysaccharide
Lu et al. NLRP 3 inflammasome may regulate inflammatory response of human periodontal ligament fibroblasts in an apoptosis‐associated speck‐like protein containing a CARD (ASC)‐dependent manner
Andrukhov et al. Response of human periodontal ligament stem cells to IFN-γ and TLR-agonists
Richert et al. A critical analysis of research methods and experimental models to study pulpitis
Chatzivasileiou et al. Dental follicle progenitor cells responses to P orphyromonas gingivalis LPS
Nokhbehsaim et al. Effects of enamel matrix derivative on periodontal wound healing in an inflammatory environment in vitro
Marshall et al. Proteomic mapping of proteins released during necrosis and apoptosis from cultured neonatal cardiac myocytes
Behm et al. Response of human mesenchymal stromal cells from periodontal tissue to LPS depends on the purity but not on the LPS source
Behm et al. Soluble CD14 enhances the response of periodontal ligament stem cells to toll‐like receptor 2 agonists
Kim et al. Aggregatibacter actinomycetemcomitans‐induced AIM2 inflammasome activation is suppressed by xylitol in differentiated THP‐1 macrophages
Varanat et al. Activation of the TREM‐1 pathway in human monocytes by periodontal pathogens and oral commensal bacteria
Zhang et al. Long non‐coding RNA lnc‐DC in dendritic cells regulates trophoblast invasion via p‐STAT3‐mediated TIMP/MMP expression
Domon et al. Age‐related alterations in gene expression of gingival fibroblasts stimulated with P orphyromonas gingivalis
Lappin et al. The influence of glycated hemoglobin on the cross susceptibility between type 1 diabetes mellitus and periodontal disease
Ohno et al. Angiopoietin-like protein 2 regulates Porphyromonas gingivalis lipopolysaccharide-induced inflammatory response in human gingival epithelial cells
Song et al. Filifactor alocis‐derived extracellular vesicles inhibit osteogenesis through TLR2 signaling
Gualtero et al. Rosuvastatin inhibits interleukin (IL)‐8 and IL‐6 production in human coronary artery endothelial cells stimulated with Aggregatibacter actinomycetemcomitans serotype b
Guo et al. CD24 activates the NLRP3 inflammasome through c‐Src kinase activity in a model of the lining epithelium of inflamed periodontal tissues