Nothing Special   »   [go: up one dir, main page]

Reddy, 2009 - Google Patents

Amyloid beta, mitochondrial structural and functional dynamics in Alzheimer's disease

Reddy, 2009

View PDF
Document ID
6352983007932283780
Author
Reddy P
Publication year
Publication venue
Experimental neurology

External Links

Snippet

Mitochondria are the major source of energy for the normal functioning of brain cells. Increasing evidence suggests that the amyloid precursor protein (APP) and amyloid beta (Aβ) accumulate in mitochondrial membranes, cause mitochondrial structural and functional …
Continue reading at pmc.ncbi.nlm.nih.gov (PDF) (other versions)

Classifications

    • GPHYSICS
    • G01MEASURING; TESTING
    • G01NINVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
    • G01N33/00Investigating or analysing materials by specific methods not covered by the preceding groups
    • G01N33/48Investigating or analysing materials by specific methods not covered by the preceding groups biological material, e.g. blood, urine; Haemocytometers
    • G01N33/50Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing
    • G01N33/68Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving proteins, peptides or amino acids
    • G01N33/6893Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving proteins, peptides or amino acids related to diseases not provided for elsewhere
    • G01N33/6896Neurological disorders, e.g. Alzheimer's disease
    • GPHYSICS
    • G01MEASURING; TESTING
    • G01NINVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
    • G01N2800/00Detection or diagnosis of diseases
    • G01N2800/28Neurological disorders
    • G01N2800/2814Dementia; Cognitive disorders
    • G01N2800/2821Alzheimer
    • GPHYSICS
    • G01MEASURING; TESTING
    • G01NINVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
    • G01N33/00Investigating or analysing materials by specific methods not covered by the preceding groups
    • G01N33/48Investigating or analysing materials by specific methods not covered by the preceding groups biological material, e.g. blood, urine; Haemocytometers
    • G01N33/50Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing
    • G01N33/5005Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving human or animal cells

Similar Documents

Publication Publication Date Title
Reddy Amyloid beta, mitochondrial structural and functional dynamics in Alzheimer's disease
Hou et al. Autophagy in Parkinson's disease
Cortes et al. TFEB dysregulation as a driver of autophagy dysfunction in neurodegenerative disease: Molecular mechanisms, cellular processes, and emerging therapeutic opportunities
Zare-Shahabadi et al. Autophagy in Alzheimer’s disease
Pérez et al. Contribution of tau pathology to mitochondrial impairment in neurodegeneration
Li et al. Endoplasmic reticulum dysfunction in Alzheimer’s disease
Ferreiro et al. Mitochondrial‐and Endoplasmic Reticulum‐Associated Oxidative Stress in Alzheimer′ s Disease: From Pathogenesis to Biomarkers
García-Escudero et al. Deconstructing mitochondrial dysfunction in Alzheimer disease
Xie et al. Ebselen ameliorates β-amyloid pathology, tau pathology, and cognitive impairment in triple-transgenic Alzheimer’s disease mice
Picone et al. Mitochondrial dysfunction: different routes to Alzheimer’s disease therapy
Canobbio et al. Role of amyloid peptides in vascular dysfunction and platelet dysregulation in Alzheimer’s disease
Reddy et al. Abnormal mitochondrial dynamics and synaptic degeneration as early events in Alzheimer's disease: implications to mitochondria-targeted antioxidant therapeutics
Reddy et al. Amyloid-β and mitochondria in aging and Alzheimer's disease: implications for synaptic damage and cognitive decline
Claeysen et al. Alzheimer culprits: cellular crossroads and interplay
Moreira et al. The key role of mitochondria in Alzheimer's disease
Dolan et al. The role of tau kinases in Alzheimer’s disease
Piaceri et al. Mitochondria and Alzheimer's disease
Zhang et al. Salidroside reduces tau hyperphosphorylation via up-regulating GSK-3β phosphorylation in a tau transgenic Drosophila model of Alzheimer’s disease
Bjørklund et al. Molecular targets in Alzheimer’s disease
Bhounsule et al. Cyclin dependent kinase 5: A novel avenue for Alzheimer’s disease
Takashima GSK-3β and memory formation
Pająk et al. Killing me softly: connotations to unfolded protein response and oxidative stress in Alzheimer’s disease
Chatterjee et al. Insulin-mediated changes in tau hyperphosphorylation and autophagy in a Drosophila model of tauopathy and neuroblastoma cells
Kim et al. The role of cell type-specific mitochondrial dysfunction in the pathogenesis of Alzheimer’s disease
Wang et al. Hypoxia‐triggered m‐Calpain activation evokes endoplasmic reticulum stress and neuropathogenesis in a transgenic mouse model of Alzheimer's disease