The GluR5 subtype of kainate receptor regulates excitatory synaptic transmission in areas CA1 and CA3 of the rat hippocampus. (Q48331477)

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7 December 2019

title

The GluR5 subtype of kainate receptor regulates excitatory synaptic transmission in areas CA1 and CA3 of the rat hippocampus (English)

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7 December 2019

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7 December 2019

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7 December 2019

V R Clarke

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7 December 2019

M J Parry

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7 December 2019

D Bleakman

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7 December 2019

D Lodge

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7 December 2019

G L Collingridge

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7 December 2019

publication date

1 October 1998

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7 December 2019

number of pages

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based on heuristic

inferred from page(s)

published in

Neuropharmacology

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7 December 2019

volume

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7 December 2019

issue

10-11

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7 December 2019

page(s)

1269-1277

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The primary afferent depolarizing action of kainate in the rat.

7 December 2019

cites work

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Cloning of a novel glutamate receptor subunit, GluR5: expression in the nervous system during development

7 January 2021

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Review: neurotransmitter receptors. II. AMPA and kainate receptors

7 January 2021

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Activity of 2,3-benzodiazepines at native rat and recombinant human glutamate receptors in vitro: stereospecificity and selectivity profiles

7 January 2021

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Kainate receptors mediate a slow postsynaptic current in hippocampal CA3 neurons

7 January 2021

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Regulation of glutamate release by presynaptic kainate receptors in the hippocampus

7 January 2021

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A hippocampal GluR5 kainate receptor regulating inhibitory synaptic transmission

7 January 2021

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GYKI 52466, a 2,3-benzodiazepine, is a highly selective, noncompetitive antagonist of AMPA/kainate receptor responses

7 January 2021

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Synaptic localization of kainic acid binding sites

7 January 2021

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Kainate-binding proteins: phylogeny, structures and possible functions

7 January 2021

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Effects of folic and kainic acids on synaptic responses of hippocampal neurones

7 January 2021

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Ibotenic acid analogues. Synthesis, molecular flexibility, and in vitro activity of agonists and antagonists at central glutamic acid receptors

7 January 2021

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The antagonism of amino acid-induced excitation of spinal neurones in the cat

7 January 2021

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The distribution of [3H]kainic acid binding sites in rat CNS as determined by autoradiography

7 January 2021

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L-[3H]Glutamate binds to kainate-, NMDA- and AMPA-sensitive binding sites: an autoradiographic analysis

7 January 2021

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Altered synaptic physiology and reduced susceptibility to kainate-induced seizures in GluR6-deficient mice

7 January 2021

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Intraventricular kainic acid preferentially destroys hippocampal pyramidal cells

7 January 2021

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Cyclothiazide reverses AMPA receptor antagonism of the 2,3-benzodiazepine, GYKI 53655

7 January 2021

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Selective antagonism of AMPA receptors unmasks kainate receptor-mediated responses in hippocampal neurons

7 January 2021

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Kainate binding sites in the hippocampal mossy fibers: Localization and plasticity

7 January 2021

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Kainic acid produces depolarization of CA3 pyramidal cells in the vitro hippocampal slice

7 January 2021

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Kainate receptors presynaptically downregulate GABAergic inhibition in the rat hippocampus.

7 January 2021

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Kainate receptor modulation of GABA release involves a metabotropic function

7 January 2021

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Distinct short-term plasticity at two excitatory synapses in the hippocampus

7 January 2021

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Inhibitors of high-affinity uptake augment depolarizations of hippocampal neurons induced by glutamate, kainate and related compounds

7 January 2021

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On the relationship between kainic acid-induced epileptiform activity and hippocampal neuronal damage

7 January 2021

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The synaptic activation of kainate receptors

7 January 2021

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The synaptic activation of the GluR5 subtype of kainate receptor in area CA3 of the rat hippocampus

7 January 2021

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Excitatory Amino Acid Transmitters

7 January 2021

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Cellular and synaptic basis of kainic acid-induced hippocampal epileptiform activity

7 January 2021

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A complex mosaic of high-affinity kainate receptors in rat brain

7 January 2021

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10.1016/S0028-3908(98)00148-8

7 January 2021

Identifiers

DOI

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7 December 2019

1 reference

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