Pages that link to "Q58883549"
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The following pages link to FLICE Induced Apoptosis in a Cell-free System (Q58883549):
Displaying 50 items.
- Negative regulation of Fas-mediated apoptosis by FAP-1 in human cancer cells (Q22254697) (← links)
- A20 inhibits tumor necrosis factor (TNF) alpha-induced apoptosis by disrupting recruitment of TRADD and RIP to the TNF receptor 1 complex in Jurkat T cells (Q24304048) (← links)
- Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors (Q24310559) (← links)
- DFF, a heterodimeric protein that functions downstream of caspase-3 to trigger DNA fragmentation during apoptosis (Q24313625) (← links)
- p28 Bap31, a Bcl-2/Bcl-XL- and Procaspase-8–associated Protein in the Endoplasmic Reticulum (Q24318875) (← links)
- The c-IAP-1 and c-IAP-2 proteins are direct inhibitors of specific caspases (Q24532851) (← links)
- IAPs block apoptotic events induced by caspase-8 and cytochrome c by direct inhibition of distinct caspases (Q24533214) (← links)
- Caspases are activated in a branched protease cascade and control distinct downstream processes in Fas-induced apoptosis (Q24647389) (← links)
- The central executioner of apoptosis: multiple connections between protease activation and mitochondria in Fas/APO-1/CD95- and ceramide-induced apoptosis (Q24653321) (← links)
- Styrylpyrone Derivative (SPD) induces apoptosis in a caspase-7-dependent manner in the human breast cancer cell line MCF-7 (Q24805678) (← links)
- HIV-1 protease processes procaspase 8 to cause mitochondrial release of cytochrome c, caspase cleavage and nuclear fragmentation (Q28211642) (← links)
- Regulation of IL-1beta generation by Pseudo-ICE and ICEBERG, two dominant negative caspase recruitment domain proteins (Q28215242) (← links)
- Apoptosis timeline (Q28215321) (← links)
- Caspase-2 induces apoptosis by releasing proapoptotic proteins from mitochondria (Q28217828) (← links)
- Biochemical Characteristics of Caspases-3, -6, -7, and -8 (Q28250824) (← links)
- ERICE, a novel FLICE-activatable caspase (Q28273537) (← links)
- Identification and functional characterization of DR6, a novel death domain-containing TNF receptor (Q28280342) (← links)
- Essential requirement for caspase-8/FLICE in the initiation of the Fas-induced apoptotic cascade (Q28282773) (← links)
- Bruton's tyrosine kinase (BTK) as a dual-function regulator of apoptosis (Q28283574) (← links)
- Two CD95 (APO-1/Fas) signaling pathways (Q29619631) (← links)
- Apoptotic response of HL-60 human leukemia cells to the antitumor drug NB-506, a glycosylated indolocarbazole inhibitor of topoisomerase 1. (Q31923228) (← links)
- Activation of the caspase cascade during Stx1-induced apoptosis in Burkitt's lymphoma cells (Q31948409) (← links)
- Ultraviolet Light Induces Apoptosis via Direct Activation of CD95 (Fas/APO-1) Independently of Its Ligand CD95L (Q32152397) (← links)
- Retinoid-dependent growth inhibition, differentiation and apoptosis in acute promyelocytic leukemia cells. Expression and activation of caspases (Q33180331) (← links)
- Proteolysis of the mismatch repair protein MLH1 by caspase-3 promotes DNA damage-induced apoptosis (Q33201671) (← links)
- Association of active caspase 8 with the mitochondrial membrane during apoptosis: potential roles in cleaving BAP31 and caspase 3 and mediating mitochondrion-endoplasmic reticulum cross talk in etoposide-induced cell death (Q33204755) (← links)
- Tumor necrosis factor alpha sensitizes malignant cells to chemotherapeutic drugs via the mitochondrial apoptosis pathway independently of caspase-8 and NF-kappaB. (Q33205160) (← links)
- Focal cerebral ischemia in the TNFalpha-transgenic rat. (Q33378976) (← links)
- Viral modulation of the host response via crmA/SPI-2 expression. (Q33653453) (← links)
- T cell signaling: a decision of life and death (Q33711156) (← links)
- Toxic effect on tissues and differentially expressed genes in hepatopancreas identified by suppression subtractive hybridization of freshwater pearl mussel (Hyriopsis cumingii) following microcystin‐LR challenge (Q33722289) (← links)
- Caspase-10-dependent cell death in Fas/CD95 signalling is not abrogated by caspase inhibitor zVAD-fmk (Q33737677) (← links)
- Caspase activation: the induced-proximity model (Q33740126) (← links)
- Selective regulation of apoptosis: the cytotoxic lymphocyte serpin proteinase inhibitor 9 protects against granzyme B-mediated apoptosis without perturbing the Fas cell death pathway (Q33781431) (← links)
- The IRF-3 transcription factor mediates Sendai virus-induced apoptosis (Q33802650) (← links)
- Cif (Cytochrome c efflux-inducing factor) activity is regulated by Bcl-2 and caspases and correlates with the activation of Bid. (Q33869177) (← links)
- Human rabaptin-5 is selectively cleaved by caspase-3 during apoptosis. (Q33884548) (← links)
- Brain ischemia and reperfusion: molecular mechanisms of neuronal injury (Q33923323) (← links)
- Proteases for cell suicide: functions and regulation of caspases (Q33927179) (← links)
- Mechanisms underlying hypoxia-induced neuronal apoptosis (Q33935219) (← links)
- Apoptosis induced by Staphylococcus aureus in epithelial cells utilizes a mechanism involving caspases 8 and 3. (Q34004467) (← links)
- Efficacy of some non-conventional herbal medications (sulforaphane, tanshinone IIA, and tetramethylpyrazine) in inducing neuroprotection in comparison with interleukin-10 after spinal cord injury: A meta-analysis. (Q34041359) (← links)
- Poxvirus homologues of cellular genes (Q34055804) (← links)
- Cell death control in lymphocytes. (Q34082790) (← links)
- Death and survival signals determine active/inactive conformations of pro-apoptotic BAX, BAD, and BID molecules (Q34170796) (← links)
- Apoptosis in amyotrophic lateral sclerosis: a review of the evidence. (Q34352949) (← links)
- Clustering of the neural cell adhesion molecule (NCAM) at the neuronal cell surface induces caspase-8- and -3-dependent changes of the spectrin meshwork required for NCAM-mediated neurite outgrowth (Q34439094) (← links)
- Proteases in cardiometabolic diseases: Pathophysiology, molecular mechanisms and clinical applications (Q34476390) (← links)
- Signaling to gene activation and cell death by tumor necrosis factor receptors and Fas. (Q34563012) (← links)
- Identification of novel compounds that enhance colon cancer cell sensitivity to inflammatory apoptotic ligands (Q34573226) (← links)