CASE REPORT - Docx Formative Case - Docxxxxxxxxx
CASE REPORT - Docx Formative Case - Docxxxxxxxxx
CASE REPORT - Docx Formative Case - Docxxxxxxxxx
14April 2019
Patient Details:
73yrs, Female, South Asian
11/04/2019
Presenting Symptoms:
Referred to tertiary eye care hospital by ophthalmologist for OCT.
Sensitivity to light
General Health:
History of contact skin allergy one month ago: 3 injections in a week, hydrocortisol cream with
cold compression.
Ocular History:
LF cataract extraction with foldable IOl implant
Clinical Signs:
Vision – OD (sc) 2/60 OS (sc) 1/60
AHP: Chin up
PD: 58mm
VD: 12.00
RT LF
NAD AC NAD
Differential Diagnosis:
Myogenic Ptosis- Malfunction of the levator muscle restricts the eyelid from being elevated into
right position. The frequent cause is myasthenia gravis, congenital myopathies, myotonic
dystrophy, and chronic progressive external ophthalmoplegia. It`s a progressive condition
which may recurrent. Variable ptosis is a key feature of Myasthenia Gravis. Ocular motility
imbalance is also an association in it that in this case is normal. The other symptoms and signs
of Myasthenia are absent (Finsterer, 2003), ( Wong , Beckingsale ,Oley and Sullivan, 2002),
(Kasnki, 2016).
Neurogenic ptosis: Ptosis may be caused due to the malfunction or disruptionof the central
nervous system or to the oculomotor orsympathetic nerve(s) .As the levator has the key role in
keeping the lid open, profound deficits of third nerve function usually results in severe or
complete ptosis.There are no signs of Third nerve here except ptosis. Diabetes, Multiple
Sclerosis also cause ptosis but the patient has no systemic disease history (Finsterer, 2003)
Mechanical ptosis: Orbital fat prolapse, eyelid tumour may cause lid to be too heavy for the
muscle to elevate it.There`s no presence of mass (Finsterer, 2003).Choroidal Revascularization
(CNV): Sudden deterioration of vision within few days especially central vision including color
confusion and metamorphosis (condition in which straight lines presume as wavy) Disruption of
new blood vessels accelerate the symptoms. (“Choroidal neovascularization”, 2019).
Cystoid Macular Edema: Retinal edema causes decrease in visual acuity along with low contrast
sensitivity and distorted lines that cause trouble in face recognition and fine vision. Patient
observes spot due to Scotia formation.( Nussenblatt ,Kaufman , Palestine & Davis
,Ferris,1987).
Retinal Detachment: It`s more common in people above the age 50.It’s a painless condition. But almost
every time the alarming signs pop-up before it occur or has advanced, as in the sudden onset of
floaters, little specks which seem to loft through field of view, Flashes of light (photopsia), Hazy view,
Progressive loss of side (peripheral) view, A curtain-like shade over field of view.
(Kasnki, 2016).
ARMD: OU
VMT: RT
Aponeurotic Ptosis: OU
Investigation:
Due to the presence scar in the left eye and flattening of macula observed through the slit lamp
it was decided to go for the OCT scan of bilateral macula to get the clear picture of fundus
pathology.It stated that view of both eyes were hazy with viterous macular traction in the right
eye. Thickness of right macula was 179um and 365um of the left. Whereas, the inner retinal
layer and retinal pigment epithelium seemed to be normal. There was an evident scaring of the
foveal region in the both eyes with no edema and hemorrhage. Macula came out to be flat in
the left eye ruling out ARMD in both eyes. Biomicroscopy & Central 10-degree computerized
automated perimetry would be done in the next visit. (“Care of patient”).
Vit.A : Vitamin A
Phaco: Phacoemulsification