Oxidative Stress and Antioxidants in Human Diseases
A special issue of International Journal of Molecular Sciences (ISSN 1422-0067). This special issue belongs to the section "Molecular Pathology, Diagnostics, and Therapeutics".
Deadline for manuscript submissions: closed (20 August 2024) | Viewed by 31660
Special Issue Editor
Interests: oxidative stress; metabolic stress; diseases; aging; antioxidants; free radical pathology; reactive oxygen species; cellular damage; antioxidant therapy
Special Issue Information
Dear Colleagues,
Since the middle of the 20th century, there has been significant progress in research on free radical biology and medicine, and accordingly, the list of diseases associated with free radicals has expanded significantly. Currently, the problem of the molecular and cellular mechanisms of oxidative stress development is extremely relevant for medical science. It was proved that excess accumulation of oxidative stress products changes proteins, fat, carbohydrates, nucleic acids, water, and electrolyte metabolism, which can cause severe tissue damages and organism adaptive capacity reduction. It can play an important role in the pathogenesis of a significant number of pathologies (obesity, diabetes mellitus, cardiovascular diseases, reproductive disorders, COVID-19, etc.), and there are also several data about the possible role of oxidative stress in the aging process. While there are widely used markers of oxidative stress, new data are emerging on other potential biomarkers for free radical damage to cells. To date, however, there is no single view on the mechanisms of free radical reactions and the antioxidant defense system regulation.
Because of that, this issue of the International Journal of Molecular Sciences will focus on new insights into the role of oxidative stress and antioxidants in the pathogenesis of human diseases.
Prof. Dr. Lyubov Iljinichna Kolesnikova
Guest Editor
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Keywords
- oxidative stress
- metabolic stress
- diseases
- aging
- antioxidants
- free radical pathology
- reactive oxygen species
- cellular damage
- antioxidant therapy
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