Nothing Special   »   [go: up one dir, main page]

Sheerin et al., 1997 - Google Patents

Protection against anti-glomerular basement membrane (GBM)-mediated nephritis in C3-and C4-deficient mice

Sheerin et al., 1997

View PDF
Document ID
5609661667015427175
Author
Sheerin N
Springall T
Carroll M
Hartley B
Sacks S
Publication year
Publication venue
Clinical & Experimental Immunology

External Links

Snippet

Mice rendered completely deficient of the complement components C3 or C4 were used to determine the influence of complement activation in the heterologous phase of the anti-GBM disease model. In wild-type animals the disease is characterized by a neutrophil infiltrate …
Continue reading at www.ncbi.nlm.nih.gov (PDF) (other versions)

Classifications

    • G—PHYSICS
    • G01—MEASURING; TESTING
    • G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
    • G01N33/00—Investigating or analysing materials by specific methods not covered by the preceding groups
    • G01N33/48—Investigating or analysing materials by specific methods not covered by the preceding groups biological material, e.g. blood, urine; Haemocytometers
    • G01N33/50—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing
    • G01N33/5005—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving human or animal cells
    • G01N33/5008—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving human or animal cells for testing or evaluating the effect of chemical or biological compounds, e.g. drugs, cosmetics
    • G—PHYSICS
    • G01—MEASURING; TESTING
    • G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
    • G01N33/00—Investigating or analysing materials by specific methods not covered by the preceding groups
    • G01N33/48—Investigating or analysing materials by specific methods not covered by the preceding groups biological material, e.g. blood, urine; Haemocytometers
    • G01N33/50—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing
    • G01N33/68—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving proteins, peptides or amino acids
    • C—CHEMISTRY; METALLURGY
    • C07—ORGANIC CHEMISTRY
    • C07K—PEPTIDES
    • C07K16/00—Immunoglobulins [IGs], e.g. monoclonal or polyclonal antibodies
    • C07K16/18—Immunoglobulins [IGs], e.g. monoclonal or polyclonal antibodies against material from animals or humans
    • C07K16/28—Immunoglobulins [IGs], e.g. monoclonal or polyclonal antibodies against material from animals or humans against receptors, cell surface antigens or cell surface determinants
    • A—HUMAN NECESSITIES
    • A01—AGRICULTURE; FORESTRY; ANIMAL HUSBANDRY; HUNTING; TRAPPING; FISHING
    • A01K—ANIMAL HUSBANDRY; CARE OF BIRDS, FISHES, INSECTS; FISHING; REARING OR BREEDING ANIMALS, NOT OTHERWISE PROVIDED FOR; NEW BREEDS OF ANIMALS
    • A01K67/00—Rearing or breeding animals, not otherwise provided for; New breeds of animals
    • A01K67/027—New breeds of vertebrates

Similar Documents

Publication Publication Date Title
Sheerin et al. Protection against anti-glomerular basement membrane (GBM)-mediated nephritis in C3-and C4-deficient mice
Tipping et al. T lymphocyte participation in antibody-induced experimental glomerulonephritis
Vogt et al. Interaction of cationized antigen with rat glomerular basement membrane: In situ immune complex formation
Kyriakides et al. Skeletal muscle reperfusion injury is mediated by neutrophils and the complement membrane attack complex
Borvak et al. Functional expression of the MHC class I-related receptor, FcRn, in endothelial cells of mice.
Johnson et al. Platelet-complement interactions in mesangial proliferative nephritis in the rat.
Olsson et al. Platelet homeostasis is regulated by platelet expression of CD47 under normal conditions and in passive immune thrombocytopenia
Frossard et al. The role of intercellular adhesion molecule 1 and neutrophils in acute pancreatitis and pancreatitis-associated lung injury
Vassalli et al. The pathogenic role of the coagulation process in rabbit Masugi nephritis
Salant et al. A new role for complement in experimental membranous nephropathy in rats.
Anniss et al. Expression of CD47 (integrin-associated protein) decreases on red blood cells during storage
Marks et al. Increased nicotinic acetylcholine receptor protein underlies chronic nicotine-induced up-regulation of nicotinic agonist binding sites in mouse brain
Panzer et al. IgM exacerbates glomerular disease progression in complement-induced glomerulopathy
Gillmore et al. Autoimmunity and glomerulonephritis in mice with targeted deletion of the serum amyloid P component gene: SAP deficiency or strain combination?
Canellini et al. Red blood cell microparticles and blood group antigens: an analysis by flow cytometry
JP2003507013A (en) Method for inhibiting the binding of β-sheet fibril to RAGE and its result
Yaar et al. The Goodpasture-like syndrome in mice induced by intravenous injections of anti-type IV collagen and anti-laminin antibody.
Chen et al. Inhibition of intravascular thrombosis in murine endotoxemia by targeted expression of hirudin and tissue factor pathway inhibitor analogs to activated endothelium
Ronco et al. Target antigens and nephritogenic antibodies in membranous nephropathy: of rats and men
Gonzalez et al. B cell acquisition of antigen in vivo
Harris et al. Serum C-reactive protein: a useful and economical marker of immune activation in renal transplantation
Castaman et al. Effectiveness of high‐dose intravenous immunoglobulin in a case of acquired von Willebrand syndrome with chronic melena not responsive to desmopressin and factor VIII concentrate
Hancock et al. Immunopathogenesis of accelerated allograft rejection in sensitized recipients: humoral and nonhumoral mechanisms1
Lim et al. Activation of mouse protease‐activated receptor‐2 induces lymphocyte adhesion and generation of reactive oxygen species
DUIJVESTIJN et al. Chronic renal allograft rejection: selective involvement of the glomerular endothelium in humoral immune reactivity and intravascular coagulation