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Aberrant regulation of human intestinal proglucagon gene expression in the NCI-H716 cell line

Endocrinology. 2003 May;144(5):2025-33. doi: 10.1210/en.2002-0049.

Abstract

Despite interest in understanding glucagon-like peptide-1 (GLP-1) production, the factors important for GLP-1 biosynthesis remain poorly understood. We examined control of human proglucagon gene expression in NCI-H716 cells, a cell line that secretes GLP-1 in a regulated manner. Insulin, phorbol myristate acetate, or forskolin, known regulators of rodent proglucagon gene expression, had no effect, whereas sodium butyrate decreased levels of NCI-H716 proglucagon mRNA transcripts. The inhibitory effect of sodium butyrate was mimicked by trichostatin A but was not detected with sodium acetate or isobutyrate. The actions of butyrate were not diminished by the ERK1/2 inhibitor PD98059, p38 inhibitor SB203580, or soluble guanylate cyclase inhibitor LY83583 or following treatment of cells with KT5823, a selective inhibitor of cGMP-dependent protein kinase. NCI-H716 cells expressed multiple proglucagon gene transcription factors including isl-1, pax-6, pax-2, cdx-2/3, pax-4, hepatocyte nuclear factor (HNF)-3 alpha, HNF-3beta, HNF-3 gamma, and Nkx2.2. Nevertheless, the butyrate-dependent inhibition of proglucagon gene expression was not associated with coordinate changes in transcription factor expression and both the human and rat transfected proglucagon promoters were transcriptionally inactive in NCI-H716 cells. Hence, NCI-H716 cells may not be a physiologically optimal model for studies of human enteroendocrine proglucagon gene transcription.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenocarcinoma / genetics*
  • Adenocarcinoma / metabolism
  • Animals
  • Butyrates / pharmacology
  • Cecal Neoplasms / genetics*
  • Cecal Neoplasms / metabolism
  • Gene Expression / drug effects
  • Gene Expression Regulation / physiology*
  • Glucagon / genetics*
  • Glucagon / metabolism
  • Glucagon-Like Peptide 1
  • Homeobox Protein Nkx-2.2
  • Homeodomain Proteins
  • Humans
  • Intestines / physiopathology*
  • Nuclear Proteins
  • Peptide Fragments / metabolism
  • Proglucagon
  • Protein Precursors / genetics*
  • Protein Precursors / metabolism
  • Rats
  • Transcription Factors / metabolism
  • Tumor Cells, Cultured

Substances

  • Butyrates
  • Homeobox Protein Nkx-2.2
  • Homeodomain Proteins
  • NKX2-2 protein, human
  • Nkx2-2 protein, rat
  • Nuclear Proteins
  • Peptide Fragments
  • Protein Precursors
  • Transcription Factors
  • Proglucagon
  • Glucagon-Like Peptide 1
  • Glucagon